时间 | 题目 | 讲者 |
---|---|---|
13:30-13:45 |
UBE2S通过招募USP15去泛素化TBK1抑制I型干扰素产生 |
翁长江 |
Changjiang Weng | ||
13:45-14:00 |
Mitochondrial STAT3-methionine metabolism axis promotes ILC2-driven allergic lung inflammation |
郭晓欢 |
Mitochondrial STAT3-methionine metabolism axis promotes ILC2-driven allergic lung inflammation |
Xiaohuan Guo | |
14:00-14:10 |
HOIL1 cleavage modulates canonical NF-kappaB signaling and inflammatory responsiveness |
冯善宇 |
HOIL1 cleavage modulates canonical NF-kappaB signaling and inflammatory responsiveness |
Shanyu Feng | |
14:10-14:20 |
The ACE2-Ang-(1-7)-Mas Axis Modulates M1/M2 Macrophage Polarization to Relieve CLP-Induced Inflammation via TLR4-Mediated NF-кb and MAPK Pathways |
潘航 |
The ACE2-Ang-(1-7)-Mas Axis Modulates M1/M2 Macrophage Polarization to Relieve CLP-Induced Inflammation via TLR4-Mediated NF-кb and MAPK Pathways |
Hang Pan | |
14:20-14:30 |
Identification of an IKKβ inhibitor for inhibition of inflammation in vivo and in vitro |
陈琪 |
Identification of an IKKβ inhibitor for inhibition of inflammation in vivo and in vitro |
Qi Chen | |
14:30-14:40 |
单核细胞迁移浸润加剧细粒棘球蚴感染小鼠肝纤维化 |
田凤鸣 |
Fengming Tian | ||
14:40-14:50 |
激活素A抑制IIA+中性粒细胞在小鼠皮肤感染组织的募集 |
齐妍 |
Yan Qi |